Shunt: Opposite case, now you've got blood flowing where its not wanted. Also why we can do CPR.plenty of O2 left in your breath for a person to absorb. Thus, their body won't blow off anymore CO2, bringing your blood CO2 levels back up and keeping you from passing out from respiratory alkalosis. On a side note, this relates into why people inhale their air when they hyperventilate right? There's plenty of O2 in the air but that air is already equilibrated with respect to CO2. Thus you get hypercarbia but no hypoxemia. Normal air has no CO2, so when that air is equilibrated its done.no more CO2 is gettin in there. Not so with CO2, which is equilibrating the other way. So there's still enough O2 left to saturate that extra blood. Remember that there's still usually a ton of residual O2 left in air you expire.a person can breathe on the air they expire. Your O2 sat will still be okay because your blood is gonna pull that O2 from the air until it equilibrates. Now all your blood is equilibrating with only half the air volume its used to. Since your blood flow is non existent for half your lung, all that blood has to go past your good alveoli. ALL your alveoli are getting perfused at 5L/min BUT only 2.5L/min are participating in gas exchange, so its basically as if you were inspiring 2.5L/min. Let's say normal ventilation is 5L/min and we'll put none of that in anatomic dead space for simplicity sake. Lets say half your alevoli are dead space because of some type of perfusion interruption. For instance, lets take a simple example. What this means is that you're only getting half as much air into exchange areas as you usually would. If you want it broken down totally here's my take on it:ĭead Space: Like jdh said, you're reducing your effective minute ventilation. Here's the link to the thread if you want to go there yourself (not much more to be had though). I'm stealing this from SDN, but it really helped me.
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